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       Pricelist 2010 

Monoclonal Antibodies
Antibodies against DNA Damage
Antibodies against AGEs-modified protein and AGE receptor

redcoon België GENTAUR BVBA

VAT BE0473327336

Av. de l Armee 68 B4

1040 Brussels BELGIUM

  Tel + 32 16 58 90 45 

Fax + 32 16 50 90 45


SIRET 48423788800017

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75005 Paris, France

 Tel 01 43 25 01 50

Fax 01 43 25 01 60 


52074 Aachen, Germany

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Fax +32 16 50 90 45

GENTAUR Pol Sp. Z.o.o. Ulica Ogarna 15/19B m2

80-826 GDANSK

Tel 00 48 51 760 77 08

Fax: 00 32 16 50 90 45


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 Tel 02 36 00 65 93

Fax 02 36 00 65 94

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Northern America 

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Other Countries redcoon België
0032 (0)16 41 44 07


Antibodies against AGEs-modified protein and AGE receptor
Advanced Glycation End Products.


Increasing evidence points to the adverse effects of advanced glycation end product modified proteins on aging-related human health. AGE modified proteins result from the non-enzymatic glycosylation of glucose with free protein amino groups reaction of carbohydrates via Maillard reaction, including advanced product formation following Amadori rearrangements. AGE accumulation is thought to contribute to pathological changes resulting in cataract formation, Alzheimer‘s disease, osteoarthritis, and myocardial dysfunction. Diabetes (hyperglycemia) is associated with accelerated of AGE formation.

Positive observation
Human aorta atherosclerotic lesion:

(Usage concentration 3µg/mL)

Anti-CML monoclonal antibody(NF-1G) has a ten times better sensibility than a former
Anti-CML monoclonal antibody(CMS-10).
This antibody is a monoclonal that very specific to CML and don’t recognize CEL and very useful for localyzed analysis in immunohistochemistry.
Renal proximal tubule and glomerulus in patients with diabetic nephropathy.


Result of an anti AGE antibody analysis, shows that AGE accumulates the following parts of (1) human lens (nondiabetic and noncataractous), (2) renal proximal tubules in patients with diabetic nephropathy and chronic renal failure,(3) diabetic retina, (4) peripheral nerves of diabetic neuropathy, (5) atherosclerotic lesions of arterial walls, (6)゚2-microglobulin forming amyloid fibrils in patients with hemodialysis-related amyloidosis, (7) senile plaques of patients with Alzheimer痴 disease, (8) the peritoneum of CAPD patients, (9) skin elastin in actinic elastosis, and (10) ceriod/lipofuscin deposits. These evidence suggests that AGE is deeply involved with Aging itself and chronic disease caused by an aging, and several AGE structure give emphasis to diabetes and brain disease field category. Various antibody Currently Proposed as AGE is CML,CEL, Pentosidine, Pyrraline, Imidazolone, Crossline and others.

Fluorescent /Crosslinked
Non-fluorescent / non-crosslinked

AGE receptors:


To be used for research only. DO NOT use for human gene therapy or clinical diagnosis!


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